There is evidence to support that. I’ll show you some very interesting pictures of implantation abnormalities in preeclampsia. It’s associated with diseases that usually have microvascular changes associated with them, such as diabetes, and hypertension. So maybe vascular changes to the uterus or placenta is the problem. If you have a really large placenta - I mentioned the hydatidiform mole, which is all placenta and no baby - but you could also have very big placentas with twins or very big placentas with some baby diseases. The thought here is that all of these bring increased risk of preeclampsia; the placentas are just so big that there is a relative reduction of perfusion, to support that hypothesis. There’s some pretty good evidence that there is reduced perfusion in preeclampsia. Animal models for preeclampsia are a major problem, but if you use them for hypothesis testing you can in several different species reduce blood supply to the placenta and get something that looks like preeclampsia.